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• Vol.64 No.6
• CLINICA NEUROL, 64: 422−426, 2024
• Vol.64 No.6 contents

Case Report

Transient neurological deficits in a patient with chronic subdural hematoma

Misa Ishiuchi, M.D.^1）, Yuichiro Inatomi, M.D.^1）, Rihito Yamamura, M.D.^2）3）, Makoto Nakajima, M.D.^4） and Toshiro Yonehara, M.D.^1）

¹⁾Department of Neurology, Saiseikai Kumamoto Hospital
²⁾Department of Neurosurgery, Saiseikai Kumamoto Hospital
³⁾Department of Neurosurgery, Aso Medical Center
⁴⁾Department of Neurology, Graduate School of Medical Sciences, Kumamoto University

A 62-year-old, right-handed man was diagnosed with asymptomatic bilateral chronic subdural hematomas and underwent hematoma removal on the left side only. At 1 month after surgery, he was admitted to our hospital because he began to have one or two attacks/day of apraxia of speech and dysesthesia of the right hand with a duration of approximately 5 min. The left hematoma had not re-expanded, but fluid-attenuated inversion resonance imaging showed hyperintense lesions in the sulci adjacent to the hematoma. Moreover, single-photon emission computed tomography revealed low-uptake lesions in the left cerebrum adjacent to the hematoma. Electroencephalogram showed no abnormalities, and CT angiography showed a slight deviation of the left middle cerebral arteries due to the hematoma. The attacks disappeared within 10 days, although the volume of the hematoma was unchanged. It was suggested that his transient neurological deficits were caused by cerebral ischemia related to chronic subdural hematoma.
Full Text of this Article in Japanese PDF (863K)

(CLINICA NEUROL, 64: 422−426, 2024)
key words: chronic subdural hematoma, transient neurological deficit, transient ischemic attack, epilepsy, cortical spreading depolarization

(Received: 22-Jan-24)