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  (Members Only)

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• Vol.41 No.12
• CLINICA NEUROL, 41: 1226−1228, 2001
• Vol.41 No.12 contents

The 42nd Annual Meeting of the Japanese Society of Neurology

Topics Seminar IX:
Channelopathy

Koichi Okamoto, M. D. and Yoshio Ikeda, M. D.

Department of Neurology, Gunma University School of Medicine

Recently, a variety of ion channel defects have been identified as the biological basis of certain familial epilepsies, paroxysmal movement disorders, myopathies and some degenerative disorders of central nervous system. Ion channel defects were mainly caused by genetic and autoimmune mechanisms. Here, we reviewed several channelopathies including spinocerebellar ataxia type 6, familial hemiplegic migraine, episodic ataxia type 2, familial hypokalemic periodic paralysis, congenital myotonia, malignant hyperthermia, epilepsy, Gitelman syndrome and Lambert-Eaton syndrome.

(CLINICA NEUROL, 41: 1226−1228, 2001)
key words: channelopathy, SCA6, periodic paralysis, Gitelman syndrome, Lambert-Eaton syndrome

(Received: 13-May-01)